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Ean ?SEM) Effects of EA treatment on plasma ACTH levels ( changes Effects of EA treatment on plasma ACTH levels ( changes vs baseline, n = 6 per group, mean ?SEM). EA treatment induced no significant changes of plasma ACTH in uninflamed rats. CFA-induced inflammation alone resulted in no significant changes in plasma ACTH levels. EA treatment in inflamed rats significantly increased ACTH levels
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Ean ?SEM) Effects of EA treatment on plasma ACTH levels ( changes Effects of EA treatment on plasma ACTH levels ( changes vs baseline, n = 6 per group, mean ?SEM). EA treatment induced no significant changes of plasma ACTH in uninflamed rats. CFA-induced inflammation alone resulted in no significant changes in plasma ACTH levels. EA treatment in inflamed rats significantly increased ACTH levels
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Ean ?SEM) Effects of EA treatment on plasma ACTH levels ( changes Effects of EA treatment on plasma ACTH levels ( changes vs baseline, n = 6 per group, mean ?SEM). EA treatment induced no significant changes of plasma ACTH in uninflamed rats. CFA-induced inflammation alone resulted in no significant changes in plasma ACTH levels. EA treatment in inflamed rats significantly increased ACTH levels
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In did not significantly change hind paw edema compared with that of sham EA rats plus vehicle, indicating that astressin had little effect on edema. Figure 5 shows the effects of astressin on EA-produced anti-hyperalgesia. PWL of the injected paw was significantly shorter compared to baseline while contralateral PWL did not change (data not shown). EA treatment significantly (P
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In did not significantly change hind paw edema compared with that of sham EA rats plus vehicle, indicating that astressin had little effect on edema. Figure 5 shows the effects of astressin on EA-produced anti-hyperalgesia. PWL of the injected paw was significantly shorter compared to baseline while contralateral PWL did not change (data not shown). EA treatment significantly (P
1
In did not significantly change hind paw edema compared with that of sham EA rats plus vehicle, indicating that astressin had little effect on edema. Figure 5 shows the effects of astressin on EA-produced anti-hyperalgesia. PWL of the injected paw was significantly shorter compared to baseline while contralateral PWL did not change (data not shown). EA treatment significantly (P
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Fect on this animal model in our previous study [3]. Since it is comparable to the treatment procedure but lacks therapeutic effect, we used it as sham control in this study.2.6 Immunohistochemistry Rats in experiment 4 were deeply anesthetized after EA treatment with sodium pentobarbital (60 mg/kg, i.p.) and immediately perfused transcardially with 4 paraformaldehyde (Sigma) in 0.1 M phosphate b
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Fect on this animal model in our previous study [3]. Since it is comparable to the treatment procedure but lacks therapeutic effect, we used it as sham control in this study.2.6 Immunohistochemistry Rats in experiment 4 were deeply anesthetized after EA treatment with sodium pentobarbital (60 mg/kg, i.p.) and immediately perfused transcardially with 4 paraformaldehyde (Sigma) in 0.1 M phosphate b